{"id":1708,"date":"2019-04-25T14:21:14","date_gmt":"2019-04-25T14:21:14","guid":{"rendered":"https:\/\/lam.unitedforclinicalnutrition.com\/es-ar\/?p=1708"},"modified":"2021-10-27T13:59:04","modified_gmt":"2021-10-27T13:59:04","slug":"nutricion-parenteral-en-el-paciente-con-sepsis","status":"publish","type":"post","link":"https:\/\/lam.unitedforclinicalnutrition.com\/es-ar\/nutricion-parenteral-en-el-paciente-con-sepsis\/","title":{"rendered":"NUTRICI\u00d3N PARENTERAL EN EL PACIENTE CON SEPSIS"},"content":{"rendered":"<p>La sepsis es la principal causa de muerte por infecci\u00f3n, especialmente si no es reconocida y tratada oportunamente.<\/p>\n<p>Es un s\u00edndrome cl\u00ednico que resulta de la respuesta inflamatoria desregulada a la infecci\u00f3n que conduce a disfunci\u00f3n org\u00e1nica<a class=\"bibliography-sup\" title=\"Singer M, Deutschman CS, Seymour CW, Shankar-Hari M, Annane D, Bauer M, Bellomo R, Bernard GR, Chiche J, Coopersmith CM, Hotchkiss RS, Levy MM, Marshall JC, Martin GS, Opal SM, Rubenfeld GD, van der Poll T, Vincent J, Angus DC. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3). JAMA. 2016;315(8):801- doi:10.1001\/jama.2016.0287\" href=\"#\" data-bibliography-link=\"\">1<\/a>. Se asocia con una alta morbimortalidad y es una de las razones m\u00e1s frecuentes de ingreso a la Unidad de Cuidados Intensivos (UCI)<a class=\"bibliography-sup\" title=\"Marik PE. Early Management of Severe Sepsis. Chest. 2014;145(6):1407\u201318. Available from:\u00a0http:\/\/dx.doi.org\/10.1378\/chest.13-2104\" href=\"#\" data-bibliography-link=\"\">2<\/a>.<\/p>\n<p>La sepsis complicada por disfunci\u00f3n org\u00e1nica se denomin\u00f3 sepsis severa, que podr\u00eda progresar al shock s\u00e9ptico, definido como sepsis inducida por la hipotensi\u00f3n persistente a pesar de un fluido adecuado<a class=\"bibliography-sup\" title=\"Levy MM, Fink MP, Marshall JC, Abraham E, Angus D, Cook D, et al. 2001 SCCM\/ESICM\/ACCP\/ATS\/SIS International Sepsis Definitions Conference. Intensive Care Med.2003;29(4):530\u20138. Available from:\u00a0http:\/\/dx.doi.org\/10.1007\/s00134-003-1662-x\" href=\"#\" data-bibliography-link=\"\">3<\/a>. (Ver Figura 1)<\/p>\n<p><img loading=\"lazy\" decoding=\"async\" class=\"alignnone size-medium wp-image-1567\" src=\"https:\/\/lam.unitedforclinicalnutrition.com\/es-mx\/wp-content\/uploads\/sites\/5\/2019\/04\/Sepsis-1-300x91.png\" alt=\"\" width=\"300\" height=\"91\" \/><\/p>\n<p>La sepsis ocurre cuando la liberaci\u00f3n de mediadores proinflamatorios en respuesta a una infecci\u00f3n supera los l\u00edmites del medio ambiente local, lo que conduce a una respuesta inflamatoria sist\u00e9mica; resultado de la incontrolada producci\u00f3n de mediadores proinflamatorios, llamada tormenta de citoquinas<a class=\"bibliography-sup\" title=\"Rittirsch D, Flierl MA, Ward PA. Harmful molecular mechanisms in sepsis. Nat Rev Immunol. 2008 Oct;8(10):776\u201387. Available from:\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2786961\/\" href=\"#\" data-bibliography-link=\"\">7<\/a>,<a class=\"bibliography-sup\" title=\"Parrish WR, Gallowitsch-Puerta M, Czura CJ, Tracey KJ. Experimental Therapeutic Strategies for Severe Sepsis. Ann N Y Acad Sci. 2008 Nov 1;1144(1):210\u201336. Available from:\u00a0http:\/\/dx.doi.org\/10.1196\/annals.1418.011\" href=\"#\" data-bibliography-link=\"\">8<\/a>.<\/p>\n<p><img loading=\"lazy\" decoding=\"async\" class=\"alignnone size-medium wp-image-1569\" src=\"https:\/\/lam.unitedforclinicalnutrition.com\/es-mx\/wp-content\/uploads\/sites\/5\/2019\/04\/Sepsis-2-300x164.png\" alt=\"\" width=\"300\" height=\"164\" \/><\/p>\n<p>La respuesta metab\u00f3lica est\u00e1 caracterizada por un sinn\u00famero de alteraciones metab\u00f3licas de nutrientes que cursa el paciente durante la fase s\u00e9ptica, entre los cuales podemos enlistar:<\/p>\n<p><img loading=\"lazy\" decoding=\"async\" class=\"alignnone size-medium wp-image-1571\" src=\"https:\/\/lam.unitedforclinicalnutrition.com\/es-mx\/wp-content\/uploads\/sites\/5\/2019\/04\/Sepsis-3-300x124.png\" alt=\"\" width=\"300\" height=\"124\" \/><\/p>\n<p>El soporte nutricional de estos pacientes es clave para evitar en gran medida las alteraciones antes mencionadas. La v\u00eda inicial de acceso es la\u00a0<u>enteral<\/u>\u00a0pero cuando esta no es suficiente para asegurar las necesidades cal\u00f3rico-proteicas, la indicaci\u00f3n de nutrici\u00f3n parenteral ha demostrado ser segura en un subgrupo de pacientes, tomando en cuenta algunas excepciones (en pacientes en shock s\u00e9ptico e inestabilidad hemodin\u00e1mica se recomienda retrasar el inicio del soporte nutricional especializado hasta que el paciente se encuentre en situaci\u00f3n estable)<a class=\"bibliography-sup\" title=\"Ortiz Leyba C, Montejo Gonz\u00e1lez JC, Vaquerizo Alonso C. Recomendaciones para el soporte nutricional y metab\u00f3lico especializado del paciente cr\u00edtico. Actualizaci\u00f3n. Consenso SEMICYUC-SENPE: Paciente s\u00e9ptico. Med Intensiva. 2011;35:72\u20136. Available from:\u00a0http:\/\/www.medintensiva.org\/es\/recomendaciones-el-soportenutricional-metabolico\/articulo-resumen\/S0210569111700158\/\" href=\"#\" data-bibliography-link=\"\">10<\/a>.<\/p>\n<p><strong>Requerimientos nutricionales en el paciente s\u00e9ptico:<\/strong><\/p>\n<ul>\n<li>El exceso de calor\u00edas puede ser perjudicial en la fase aguda:\n<ul>\n<li><strong>Provisi\u00f3n m\u00e1xima<\/strong>: 20 kcal\/kd\/d<\/li>\n<\/ul>\n<\/li>\n<li>Durante las fases hipermetab\u00f3lica y de recuperaci\u00f3n:\n<ul>\n<li><strong>Aporte cal\u00f3rico<\/strong>\u00a0de 25-30 kcal\/kg\/d (2\/3 de carbohidratos, 1\/3 de l\u00edpidos)<\/li>\n<li><strong>Aporte de prote\u00ednas:<\/strong>2-1.5 g\/kg\/d<\/li>\n<li><strong>Aporte de l\u00edpidos:<\/strong>\u00a01-1.5 g\/kg\/d<\/li>\n<li><strong>Relaci\u00f3n calor\u00edas no proteicas \/g de Nitrogeno de 100 a 150:1<\/strong><\/li>\n<\/ul>\n<\/li>\n<li>Importante: Supervisar y adaptar el aporte nutricional en cada caso de acuerdo a cambios en la composici\u00f3n corporal y el comportamiento.<a class=\"bibliography-sup\" title=\"Lheureux O, Preiser J-C. Role of Nutrition Support in Inflammatory Conditions . Nutr Clin Pract. SAGE Publications Inc; 2017 Mar 28;32(3):310\u20137. Available from:\u00a0http:\/\/dx.doi.org\/10.1177\/0884533617695242\" href=\"#\" data-bibliography-link=\"\">11<\/a><\/li>\n<li>Deber\u00e1 tomarse en cuenta un aporte diario de elementos traza y vitaminas, especialmente Zinc, Selenio y Vitaminas del Complejo B.<a class=\"bibliography-sup\" title=\"Gil A. Tratado de nutrici\u00f3n Tomo IV, Nutrici\u00f3n en el trauma, la sepsisy las quemaduras.Editor M\u00e9dica Panam Cap\u00edtulo. 2010;16:481\" href=\"#\" data-bibliography-link=\"\">9<\/a><\/li>\n<\/ul>\n<p><strong>Composici\u00f3n de la NP:<\/strong><\/p>\n<p>La composici\u00f3n de la NPT (Nutrici\u00f3n Parenteral Total) incluye una soluci\u00f3n de glucosa, l\u00edpidos, amino\u00e1cidos, vitaminas y minerales.<\/p>\n<p>La emulsiones de l\u00edpidos son \u00fatiles en los pacientes con intolerancia a la glucosa, al proporcionar una menor cantidad de calor\u00edas en forma de carbihidratos. En el paciente con sepsis al utilizar emulsiones con aceite de pescado, se ha observado una disminuci\u00f3n de la estancia en UCI y duraci\u00f3n de los d\u00edas de ventilaci\u00f3n mec\u00e1nica. <a class=\"bibliography-sup\" title=\"Taeb, A. M., Hooper, M. H., &amp; Marik, P. E. Sepsis: Current Definition, Pathophysiology, Diagnosis, and Management. Nutrition in Clinical Practice. 2017;32(3), 296\u2013308.\u00a0http:\/\/doi.org\/10.1177\/0884533617695243\" href=\"#\" data-bibliography-link=\"\">4<\/a><\/p>\n<p>En cuanto a los amino\u00e1cidos, tras la agresi\u00f3n, la glutamina desempe\u00f1a un papel importante en inducir mecanismos de protecci\u00f3n celular a trav\u00e9s, principalmente, del aumento en la producci\u00f3n de las porte\u00ednas de fase de choque, ya que su expresi\u00f3n protege frente al da\u00f1o celular y frente a mecanismos de isquemia\/perfusi\u00f3n,<a class=\"bibliography-sup\" title=\"Ziegler TR, Ogden LG, Singleton KD, Luo M, Fernandez-Estivariz C, Griffith DP, et al.Parenteral glutamine increases serum heat shock protein 70 in critically ill patients. Intensive Care Med. 2005;31(8):1079\u201386. Available from: http:\/\/dx.doi.org\/10.1007\/s00134-005-2690-5\" href=\"#\" data-bibliography-link=\"\">12<\/a>\u00a0lo que le confiere un papel potencial para evitar la progresi\u00f3n a da\u00f1o multiorg\u00e1nico y se podr\u00eda suplementar en los pacientes que requieren nutrici\u00f3n parenteral.<a class=\"bibliography-sup\" title=\"Gil A. Tratado de nutrici\u00f3n Tomo IV, Nutrici\u00f3n en el trauma, la sepsisy las quemaduras.Editor M\u00e9dica Panam Cap\u00edtulo. 2010;16:481\" href=\"#\" data-bibliography-link=\"\">9<\/a><\/p>\n<p><strong>Referencias bibliogr\u00e1ficas:<\/strong><\/p>\n<ol>\n<li>Singer M, Deutschman CS, Seymour CW, Shankar-Hari M, Annane D, Bauer M, Bellomo R, Bernard GR, Chiche J, Coopersmith CM, Hotchkiss RS, Levy MM, Marshall JC, Martin GS, Opal SM, Rubenfeld GD, van der Poll T, Vincent J, Angus DC. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3). JAMA. 2016;315(8):801- doi:10.1001\/jama.2016.0287<\/li>\n<li>Marik PE. Early Management of Severe Sepsis. Chest. 2014;145(6):1407\u201318. Available from:\u00a0<a href=\"http:\/\/dx.doi.org\/10.1378\/chest.13-2104\">http:\/\/dx.doi.org\/10.1378\/chest.13-2104<\/a><\/li>\n<li>Levy MM, Fink MP, Marshall JC, Abraham E, Angus D, Cook D, et al. 2001 SCCM\/ESICM\/ACCP\/ATS\/SIS International Sepsis Definitions Conference. Intensive Care Med.2003;29(4):530\u20138. Available from:\u00a0<a href=\"http:\/\/dx.doi.org\/10.1007\/s00134-003-1662-x\">http:\/\/dx.doi.org\/10.1007\/s00134-003-1662-x<\/a><\/li>\n<li>Taeb, A. M., Hooper, M. H., &amp; Marik, P. E. Sepsis: Current Definition, Pathophysiology, Diagnosis, and Management. Nutrition in Clinical Practice. 2017;32(3), 296\u2013308.\u00a0<a href=\"http:\/\/doi.org\/10.1177\/0884533617695243\">http:\/\/doi.org\/10.1177\/0884533617695243<\/a><\/li>\n<li>RC B. Immunologic dissonance: A continuing evolution in our understanding of the systemic inflammatory response syndrome (sirs) and the multiple organ dysfunction syndrome (mods). Ann Intern Med. 1996 Oct 15;125(8):680\u20137. Available from:\u00a0<a href=\"http:\/\/dx.doi.org\/10.7326\/0003-4819-125-8-199610150-00009\">http:\/\/dx.doi.org\/10.7326\/0003-4819-125-8-199610150-00009<\/a><\/li>\n<li>Schulte W, Bernhagen J, Bucala R. Cytokines in sepsis: potent immunoregulators and potential therapeutic targets\u2014an updated view. Mediators Inflamm. 2013;2013(6):165974.. doi:10.1155\/2013\/165974<\/li>\n<li>Rittirsch D, Flierl MA, Ward PA. Harmful molecular mechanisms in sepsis. Nat Rev Immunol. 2008 Oct;8(10):776\u201387. Available from:\u00a0<a href=\"http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2786961\/\">http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2786961\/<\/a><\/li>\n<li>Parrish WR, Gallowitsch-Puerta M, Czura CJ, Tracey KJ. Experimental Therapeutic Strategies for Severe Sepsis. Ann N Y Acad Sci. 2008 Nov 1;1144(1):210\u201336. Available from:\u00a0<a href=\"http:\/\/dx.doi.org\/10.1196\/annals.1418.011\">http:\/\/dx.doi.org\/10.1196\/annals.1418.011<\/a><\/li>\n<li>Gil A. Tratado de nutrici\u00f3n Tomo IV, Nutrici\u00f3n en el trauma, la sepsisy las quemaduras.Editor M\u00e9dica Panam Cap\u00edtulo. 2010;16:481<\/li>\n<li>Ortiz Leyba C, Montejo Gonz\u00e1lez JC, Vaquerizo Alonso C. Recomendaciones para el soporte nutricional y metab\u00f3lico especializado del paciente cr\u00edtico. Actualizaci\u00f3n. Consenso SEMICYUC-SENPE: Paciente s\u00e9ptico. Med Intensiva. 2011;35:72\u20136. Available from:\u00a0<a href=\"http:\/\/www.medintensiva.org\/es\/recomendaciones-el-soportenutricional-metabolico\/articulo-resumen\/S0210569111700158\/\">http:\/\/www.medintensiva.org\/es\/recomendaciones-el-soportenutricional-metabolico\/articulo-resumen\/S0210569111700158\/<\/a><\/li>\n<li>Lheureux O, Preiser J-C. Role of Nutrition Support in Inflammatory Conditions . Nutr Clin Pract. SAGE Publications Inc; 2017 Mar 28;32(3):310\u20137. Available from:\u00a0<a href=\"http:\/\/dx.doi.org\/10.1177\/0884533617695242\">http:\/\/dx.doi.org\/10.1177\/0884533617695242<\/a><\/li>\n<li>Ziegler TR, Ogden LG, Singleton KD, Luo M, Fernandez-Estivariz C, Griffith DP, et al.Parenteral glutamine increases serum heat shock protein 70 in critically ill patients. Intensive Care Med. 2005;31(8):1079\u201386. Available from: http:\/\/dx.doi.org\/10.1007\/s00134-005-2690-5<\/li>\n<\/ol>\n","protected":false},"excerpt":{"rendered":"<p>La sepsis es la principal causa de muerte por infecci\u00f3n, especialmente si no es reconocida y tratada oportunamente. Es un s\u00edndrome cl\u00ednico que resulta de la respuesta inflamatoria desregulada a la infecci\u00f3n que conduce a disfunci\u00f3n org\u00e1nica1. Se asocia con una alta morbimortalidad y es una de las razones m\u00e1s frecuentes de ingreso a la [&hellip;]<\/p>\n","protected":false},"author":3188,"featured_media":1709,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"footnotes":""},"categories":[58,60,64],"tags":[],"class_list":["post-1708","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-articulo","category-articulo-2","category-articulo-4"],"acf":[],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v24.5 - https:\/\/yoast.com\/wordpress\/plugins\/seo\/ -->\n<title>NUTRICI\u00d3N PARENTERAL EN EL PACIENTE CON SEPSIS - Unidos por la Nutrici\u00f3n Cl\u00ednica | Argentina<\/title>\n<meta name=\"description\" content=\"La sepsis es la principal causa de muerte por infecci\u00f3n, especialmente si no es reconocida y tratada oportunamente. 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